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Dr. Miranda Bader

Senior Scientist
Free Choice Enterprises, Ltd.
 

Brief Biography:


Miranda L. Bader Lange, Ph.D.

Current:

  Primary: Senior Scientist at Free Chioce Enterprises, Ltd.

  Secondary: Freelance Scientific Editor

 

Education:

1. Doctor of Philosophy (Ph.D.), Chemistry, Neurochemistry emphasis, University of Kentucky, Lexington, KY, USA (2005-2010)

2. Bachelor of Science (B.S.), Biochemistry and Molecular Biology, University of Wisconsin-Eau Claire, Eau Claire, WI, USA (2000-2005)

 

Academic positions:


- Post-Doctoral Research Fellow, Department of Neurology (2011 – 2013), Mayo Clinic, Rochester, MN, USA

- Post-Doctoral Research Fellow, Department of Laboratory Medicine & Pathology (2010 – 2011), Mayo Clinic, Rochester, MN, USA

- Freelance Science Editor (2010 – Present)

- Graduate Student Researcher (2005 – 2010), University of Kentucky, Lexington, KY, USA

- Private Tutor (2006-2009), University of Kentucky, Lexington, KY, USA

- General Chemistry Teaching Assistant (2005 – 2008), University of Kentucky, Lexington, KY, USA

- Undergraduate Student/Faculty Researcher (2004 – 2005), University of Wisconsin-Eau Claire, Eau Claire, WI, USA

 

 

Research interests:


Aging, Biochemistry, Brain, and Neuroscience

 

Any other information:


Publications: 

  1. Staff N.P., Podratz J., Grassner L., Bader M., Paz J., Knight A.M., Loprinzi C.L., Trushina E., Windebank A.J. (2013). Bortezomib alters microtubule polymerization and axonal transport in rat dorsal root ganglion neurons. Neurotoxicology 39, 124-31.
  2. Lange M., Zeng Y., Knight A., Windebank A., Trushina E. (2012). Comprehensive method for culturing embryonic dorsal root ganglion neurons for Seahorse Extracellular Flux XF24 Analysis. Front Neurol 3, 175-.
  3. Sultana R.,* Sowell Robinson R.A.,* Bader Lange M.L.,* Fiorini A., Galvan V., Fombonne J., Baker A., Gorostiza O., Zhang J., Cai J., Pierce W.M., Bredesen D.E., and Butterfield D.A. (2012). Do proteomics analyses provide insights into reduced oxidative stress in the brain of an Alzheimer disease transgenic mouse model with an M631L amyloid precursor protein substitution and thereby the importance of amyloid-beta-resident methionine 35 in Alzheimer disease pathogenesis? Antiox Redox Sig 17, 1507-14.
  4. Pocernich C.B., Bader Lange M.L., Sultana R., and Butterfield D.A. (2011).  Nutritional approaches to modulate oxidative stress in Alzheimer’s disease.  Curr Alzheimer Res 8, 452-69.
  5. Sowell Robinson R.A.,* Bader Lange M.L.,* Sultana R., Galvan V., Fombonne J., Gorostiza O., Zhang J., Warrier G., Cai J., Pierce W.M., Bredesen D.E., and Butterfield D.A. (2011).  Differential expression and redox proteomics of an Alzheimer disease transgenic mouse model:  Effects of the amyloid-β peptide of APP.  Neuroscience 177, 207-22.
  6. Bader Lange M.L., St. Clair, D., Markesbery, W.R., Studzinski, C.M., Murphy, M.P., and Butterfield, D.A. (2010).  Age-related loss of phospholipid asymmetry in APPNLh/APPNLh x PS-1P264L/PS-1P264L human double knock-in mice: Relevance to Alzheimer disease.  Neurobiol Dis 38, 104-115.
  7. Butterfield D.A., Hardas S., and Bader Lange M.L. (2010).  Oxidatively modified glyceraldehyde-3-phosphate dehydrogenase (GAPDH) and Alzheimer disease: Many pathways to neurodegeneration. J Alzheimers Dis 20, 369-93.
  8. Butterfield D.A., Bader Lange M.L., and Sultana R. (2010).  Involvements of the lipid peroxidation product, HNE, in the pathogenesis and progression of Alzheimer’s disease. Biochim Biophys Acta 1801, 924-29.
  9. ButterfieldD.A., Galvan V., Bader Lange M.L., Tang H., Sowell R.A., Fombonne J., Gorostiza O., Zhang J., Sultana R.,and Bredesen D.E. (2010).  In vivo oxidative stress in Alzheimer disease transgenic mice: Requirement for methionine 35 in amyloid β-peptide of APP. Free Radic Biol Med 48, 136-44.
  10. Butterfield D.A. and Bader Lange M.L. (2009).  The multifunctional role of enolase in Alzheimer disease: Beyond altered glucose metabolism. J Neurochem 111, 915-33.
  11. Bader Lange M.L., Cenini G., Pirrodi M., Mohmmad Abdul H., Sultana R., Galli F., Memo M., and Butterfield D.A. (2008). Loss of phospholipid asymmetry and elevated brain apoptotic protein levels in subjects with amnestic mild cognitive impairment and Alzheimer disease. Neurobiol Dis 29, 456-64.
 

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